PlantFunDestroying angel Toxic?
Symptom onset is delayed 8-12 hours (up to 48 hours) post-ingestion, with a temporary 12-24 hour asymptomatic phase before severe organ damage develops.
Amatoxins are heat-stable, so cooking, drying or freezing does not reduce toxicity, and remains active for over 10 years in dried mushroom specimens.
Even small ingestion amounts can cause fatal liver and kidney failure without prompt, targeted medical intervention, with no home remedy effective for neutralizing toxins.
FAQ
Is Destroying angel toxic to Humans?
Latent period of 8-12 hours (range 4-48 hours), followed by sudden severe gastrointestinal distress: nausea, vomiting, colicky abdominal pain, watery diarrhea, severe dehydration. A 12-24 hour asymptomatic phase follows initial symptom treatment, then the hepatorenal phase begins 2-4 days post-ingestion with elevated liver enzymes, jaundice, reduced clotting factors, bleeding, hepatic encephalopathy, hepatic coma, uremia and kidney failure. (Severe)
Is Destroying angel toxic to Cats?
No species-specific data available; suspected to cause identical severe hepatorenal toxicity as in humans, with very high risk of fatality even from small ingestion amounts. (Severe)
Is Destroying angel toxic to Dogs?
No species-specific data available; suspected to cause identical severe hepatorenal toxicity as in humans, with very high risk of fatality even from small ingestion amounts. (Severe)
Is Destroying angel toxic to Horses?
No species-specific data available; suspected to cause identical severe hepatorenal toxicity as in humans, with very high risk of fatality even from small ingestion amounts. (Severe)
Toxicity Details
Latent period of 8-12 hours (range 4-48 hours), followed by sudden severe gastrointestinal distress: nausea, vomiting, colicky abdominal pain, watery diarrhea, severe dehydration. A 12-24 hour asymptomatic phase follows initial symptom treatment, then the hepatorenal phase begins 2-4 days post-ingestion with elevated liver enzymes, jaundice, reduced clotting factors, bleeding, hepatic encephalopathy, hepatic coma, uremia and kidney failure.
No species-specific data available; suspected to cause identical severe hepatorenal toxicity as in humans, with very high risk of fatality even from small ingestion amounts.
No species-specific data available; suspected to cause identical severe hepatorenal toxicity as in humans, with very high risk of fatality even from small ingestion amounts.
No species-specific data available; suspected to cause identical severe hepatorenal toxicity as in humans, with very high risk of fatality even from small ingestion amounts.
Toxicity Snapshot
First Aid & Prevention
First 30 Minutes
- • Induce vomiting only if ingestion occurred within the past 2 hours and the affected individual/pet is fully conscious.
- • Administer activated charcoal as soon as possible to reduce toxin absorption in the gut.
- • Do not give food, water, or anti-emetic medications unless explicitly instructed by a medical/vet professional.
- • Seek immediate emergency care even if no symptoms are present, as delayed treatment drastically increases fatality risk.
Contact Vet Immediately
- • Any confirmed or suspected ingestion of this mushroom by a pet.
- • Onset of unexplained vomiting, diarrhea or abdominal pain after wild mushroom access.
- • Signs of jaundice (yellowing of gums/skin), confusion, or reduced urine output post-exposure.
Prevention
- • Only consume wild mushrooms if positively identified by a certified, experienced mushroom expert.
- • Discard any wild mushroom with ambiguous identification features, especially all-white Amanita specimens.
- • Keep all foraged wild mushrooms out of reach of children and pets at all times.
Treatment & Prognosis
- • Repetitive activated charcoal administration to interrupt enterohepatic circulation of amatoxins.
- • Administration of cytoprotective agents (cyclosporin A, rifampicin, silibinin) to block liver cell uptake of toxins.
- • N-acetylcysteine administration to reduce liver damage if elevated liver enzyme levels are detected.
- • Supportive care including fluid replacement for dehydration, coagulation support, and continuous liver/kidney function monitoring; emergency liver transplant may be required for severe human cases.
Professional Toxicology
• Amatoxins bind to RNA polymerase II in liver cells, inhibiting protein synthesis and causing widespread cell necrosis.
• Toxins undergo enterohepatic circulation, prolonging toxicity duration and increasing organ damage.
• Secondary kidney damage occurs as unmetabolized toxins are filtered through renal tissue.
Information from CliniTox veterinary text.
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Data Sources
This information is for reference only and does not constitute medical advice. If ingestion or poisoning is suspected, seek medical or veterinary attention immediately.