Northern wolfsbane Toxic?
All plant parts are toxic, especially roots, seeds and flower water; above-ground parts have the highest toxin content before flowering.
Aconitine, one of the strongest known plant poisons, makes up 0.2-3% of the plant's dry weight, with toxicity reduced slightly by drying or long storage.
Symptoms onset within minutes of ingestion, with death from respiratory paralysis or cardiac arrest possible if urgent treatment is not received.
FAQ
Is Northern wolfsbane toxic to Humans?
Burning and paresthesia in the mouth shortly after ingestion, followed by salivation, nausea, vomiting, diarrhea, thirst, colic, restlessness, anxiety, ataxia, weakness, convulsions, possible coma, hypotension, irregular heartbeat, ventricular fibrillation, dilated pupils, vision loss up to blindness, hypothermia, dyspnea, cyanosis, death from respiratory or cardiac failure. (Severe)
Is Northern wolfsbane toxic to Cats?
No species-specific symptom data available; expected to present with similar neurological, cardiac and respiratory distress as seen in human poisoning, which may be rapidly fatal. (Severe)
Is Northern wolfsbane toxic to Dogs?
No species-specific symptom data available; expected to present with similar neurological, cardiac and respiratory distress as seen in human poisoning, which may be rapidly fatal. (Severe)
Is Northern wolfsbane toxic to Horses?
Higher sensitivity to poisoning than sheep; symptoms include rapid onset of neurological, cardiac and respiratory distress, with small ingested amounts potentially causing fatal outcomes. (Severe)
Toxicity Details
Burning and paresthesia in the mouth shortly after ingestion, followed by salivation, nausea, vomiting, diarrhea, thirst, colic, restlessness, anxiety, ataxia, weakness, convulsions, possible coma, hypotension, irregular heartbeat, ventricular fibrillation, dilated pupils, vision loss up to blindness, hypothermia, dyspnea, cyanosis, death from respiratory or cardiac failure.
No species-specific symptom data available; expected to present with similar neurological, cardiac and respiratory distress as seen in human poisoning, which may be rapidly fatal.
No species-specific symptom data available; expected to present with similar neurological, cardiac and respiratory distress as seen in human poisoning, which may be rapidly fatal.
Higher sensitivity to poisoning than sheep; symptoms include rapid onset of neurological, cardiac and respiratory distress, with small ingested amounts potentially causing fatal outcomes.
Toxicity Snapshot
First Aid & Prevention
First 30 Minutes
- • If ingested and the affected individual/animal is conscious, immediately induce vomiting to remove unabsorbed plant material.
- • Administer activated charcoal to bind remaining toxins in the gastrointestinal tract.
- • Do not give any food, drink or medications unless instructed by a medical/veterinary professional.
- • Seek emergency medical or veterinary care immediately, as symptom onset is extremely rapid.
Contact Vet Immediately
- • Any confirmed or suspected ingestion of any part of Northern wolfsbane by pets or livestock.
- • Appearance of symptoms including excessive salivation, uncoordinated movement, irregular heartbeat, or difficulty breathing after possible exposure.
- • Detection of Northern wolfsbane contamination in animal feed or forage supplies.
Prevention
- • Do not cultivate Northern wolfsbane in areas accessible to children, pets or grazing livestock.
- • Always fully verify the identity of wild foraged plants before consumption, to avoid confusion with edible celery or horseradish.
- • Wear impermeable protective gloves when handling this plant to prevent skin absorption of toxic alkaloids.
Treatment & Prognosis
- • Gastrointestinal decontamination via induced emesis, gastric lavage and activated charcoal administration as clinically appropriate.
- • Symptomatic supportive care: control cardiac arrhythmias, maintain respiratory function, manage seizures, correct hypothermia and fluid/electrolyte imbalances.
- • Avoid administration of Digitalis or Strophanthin, as these are contraindicated for aconitine poisoning.
Professional Toxicology
• Aconitine and related alkaloids bind to voltage-gated sodium channels, causing persistent activation of nerve and muscle cells.
• Toxins disrupt cardiac electrical function leading to life-threatening arrhythmias, and impair neuromuscular transmission.
• Progressive untreated toxicity leads to respiratory paralysis or cardiac arrest, usually within hours of ingestion.
Information from CliniTox veterinary text.
The following plants look similar and may be confused:
Related Toxic Plants
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Data Sources
This information is for reference only and does not constitute medical advice. If ingestion or poisoning is suspected, seek medical or veterinary attention immediately.

